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Speech Motor ControlNew developments in basic and applied research$
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Ben Maassen and Pascal van Lieshout

Print publication date: 2010

Print ISBN-13: 9780199235797

Published to Oxford Scholarship Online: March 2012

DOI: 10.1093/acprof:oso/9780199235797.001.0001

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Laryngeal articulatory coupling in three speech disorders

Laryngeal articulatory coupling in three speech disorders

Chapter:
(p.283) Chapter 17 Laryngeal articulatory coupling in three speech disorders
Source:
Speech Motor Control
Author(s):

Christopher Dromey

Publisher:
Oxford University Press
DOI:10.1093/acprof:oso/9780199235797.003.0017

This chapter focuses on data from three speech disorders to illustrate articulatory changes that arise when the larynx is the nominal target of treatment. In each case, articulatory acoustic or kinematic data were collected to determine whether clinical efforts directed at phonation would have measurable effects on articulator movement. The finding of treatment effects spreading beyond the target of the larynx has important implications for our understanding of disordered speech motor control as well as for intervention strategies in these populations. Lip kinematics in seven speakers with spasmodic dysphonia were recorded before and after Botox injection of the vocal folds. Bilabial coordination indexes were more aberrant when spasm severity was greatest, moving towards control speaker values after voice treatment. Pre/post treatment recordings from 111 women with muscle tension dysphonia were analyzed for evidence of articulatory changes. Although treatment focused on the larynx, increases in vowel space and diphthong formant transitions suggest that increased articulatory dynamics accompanied the voice improvements. Lip kinematic data from ten speakers with Parkinson's disease (PD) revealed increased amplitudes and velocities, along with more consistent trajectories over multiple repetitions for loud speech. Acoustic recordings from a speaker with PD were analyzed for F2 diphthong transitions, which revealed evidence of increased articulatory excursions following intensive voice treatment, even though no therapy exercises involved articulation. The data from these speakers reveal altered vocal tract behavior when the larynx is the primary target of therapy.

Keywords:   larynx, articulation, spasmodic dysphonia, neurodegenerative disease, muscle tension dysphonia

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