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Coronary Heart Disease EpidemiologyFrom aetiology to public health$
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Michael Marmot and Paul Elliott

Print publication date: 2005

Print ISBN-13: 9780198525738

Published to Oxford Scholarship Online: September 2009

DOI: 10.1093/acprof:oso/9780198525738.001.0001

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Gene–environment interaction in coronary artery disease: apolipoprotein E and smoking and the interleukin-6 gene and inflammation as examples

Gene–environment interaction in coronary artery disease: apolipoprotein E and smoking and the interleukin-6 gene and inflammation as examples

Chapter:
(p.528) Chapter 31 Gene–environment interaction in coronary artery disease: apolipoprotein E and smoking and the interleukin-6 gene and inflammation as examples
Source:
Coronary Heart Disease Epidemiology
Author(s):

S. E. Humphries

Publisher:
Oxford University Press
DOI:10.1093/acprof:oso/9780198525738.003.0032

This chapter discusses gene-environment interaction in coronary artery disease (CAD). Topics covered include risk factors for CAD, apolipoprotein E and effect on lipid levels and coronary heart disease risk, APOE genotype-smoking interaction, homeostasis and use of stressing the genotype to identify functional variants, and the role of inflammatory processes in atherogenesis.

Keywords:   coronary artery disease, coronary heart disease, lipid levels, inflammation, candidate genes

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